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Eric Reiter

Education

➢ Ph.D. thesis, 1996 Université de Liège, Belgium, Laboratory of Pr G. Hennen.

➢ Qualification to supervise Ph.D. theses (HDR) Université de Tours, 2002.

Position

➢ Since 2006 : Directeur de Recherche INRA

➢ 1996-2006 : Chargé de Recherche INRA

➢ 1996-2003 : “Mécanismes d’action des gonadotropines” team, UMR 6175 PRC, Nouzilly.

➢ 2003-2005 : Visiting scientist in Pr Robert Lefkowitz’s laboratory, Howard Hughes Medical Institute, Duke University, Durham, NC, USA

➢ 2006-2008 : Leader of the group “Mécanismes d’action des gonadotropines” (MAG)

➢ 2009-2011 : Co-leader of the group Biology and Bioinformatics of Signaling Systems (BIOS, http://bios.tours.inra.fr)

➢ Since 2012 : Member of the BIOS group.

Professional achievements

➢ External evaluator for the ERC “starting grants”, 7th FP (2007), ESF, BBSRC and ANR.

➢ Reviewer for several international journals including Endocrinology, Mol Endocrinology, J Neurochem, JCEM, J Endo, J Mol Endo, Reprod Biol Endo, Curr Pharm Des, Biol Reprod,…

➢ Organizer of an ESF exploratory workshop on “Systems biology of GPCR signaling” Paris, November 2009 (http://www.esf.org/activities/explo...)

➢ Associate Editor of Frontiers in Cellular Endocrinology Selected personal grants

➢ AIP INRA AgroBI : 2006-2008, 100 K€ (4 partners, coordinator)

➢ ANR RIB PhosphoProteomeArray 2008-2009, 15 K€ (coordinator V. Sakanyan)

➢ AE INRIA-INRA Regate (2009-2012), 100 K€ (INRA coordinator, coordinator F. Clément)

➢ Research grant from Région Centre RASTA, 2009-2011, 206 K€ (4 partners, coordinator)

➢ “Jeune équipe” INRA BIOS 2009-2011, 90 K€ (coordinator A. Poupon)

➢ ANR blanc GPCRnet 2011-2015, 600 K€ (2 partners, coordinator)

➢ Research grant from Région Centre ARTE-2, 2012-2014, 200 K€ (2 partners, coordinator)

Publications

➢ Author more than 50 publications in international peer-reviewed journals (click here for an updated list and bibliometrical profile), 2 book chapters and more than 35 congress abstracts.

Selected publications

➢ Ren X.R. *, Reiter E.*, Ahn S., Kim J., Chen W., Lefkowitz R.J. 2005. Different G protein-coupled receptor kinases govern G protein and beta-arrestin-mediated signalling of V2 vasopressin receptor. Proc Nat Acad Sci. 102:1448-1453. *Equivalent contributions

➢ Reiter E., Lefkowitz R.J. 2006. GRKs and beta-arrestins : roles in receptor silencing, trafficking and signaling. Trends in Endocrinology and Metabolism 17 (4), 159-165.

➢ Kara E., Crepieux P., Gauthier C., Martinat N., Piketty V., Guillou F., Reiter E. 2006. A phosphorylation cluster of five serine and threonine residues in the C-terminus of the follicle-stimulating hormone receptor is important for desensitization but not for beta-arrestin-mediated ERK activation. Molecular Endocrinology 20 (11), 3014-3026.

➢ Dupuy L., Gauthier C., Durand G., Musnier A., Heitzler D., Herledan A., Sakanyan V., Crepieux P., Reiter E. 2009. A highly sensitive near-infrared fluorescent detection method to analyse signaling pathways by reverse-phase protein array. Proteomics 9 (24), 5446-5454.

➢ Wehbi V., Tranchant T., Durand G., Musnier A., Decourtye J., Piketty V., Butnev V.Y., Bousfield G.R., Crepieux P., Maurel M.C., Reiter E. 2010. Partially deglycosylated equine LH preferentially activates beta-arrestin-dependent signaling at the follicle-stimulating hormone receptor. Molecular Endocrinology 24(3), 561-573.

➢ Tranchant T., Durand G., Gauthier C., Crépieux P., Ulloa-Aguirre A., Royère D., Reiter E. 2011. Preferential β-arrestin signalling at low receptor density revealed by functional characterization of the human FSH receptor A189 V mutation. Molecular and Cellular Endocrinology 331(1):109-18.

➢ Reiter E., Ahn S., Shukla A.K., Lefkowitz R.J. 2012. Molecular Mechanism of β-Arrestin-Biased Agonism at Seven-Transmembrane Receptors. Annual Reviews in Pharmacology and Toxicology 52:179-97.

➢ Heitzler D, Durand G, Gallay N, Rizk A, Ahn S, Kim J, Violin JD, Dupuy L, Gauthier C, Piketty V, Crépieux P, Poupon A, Clément F, Fages F, Lefkowitz RJ, Reiter E. 2012. Competing G protein-coupled receptor kinases balance G protein and β-arrestin signaling. Molecular Systems Biology 8:590.

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